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Ren Y, Han X, Yu K, Sun S, Zhen L, Li Z, Wang S*. microRNA-200c downregulates XIAP expression to suppress proliferation and promote apoptosis of triple-negative breast cancer cells. Mol Med Rep, 2014, 10(1), 315-32141. Chen J, Jiang P, Wang HJ, Zhang JY, Xu Y, Guo MH, Zhang B, Tang CY, Cao HY, Wang S*. The efficacy of molecular subtyping in predicting postoperative recurrence in breast-conserving therapy: a 15-study meta-analysis. World J Surg Oncol, 2014, 12, 21242. Zhou W, Jiang Y, Chen L, Ling L, Liang M, Pan H, Wang S, Ding Q, Liu X, Wang S*. Image and pathological changes after microwave ablation of breast cancer: a pilot study. Eur J Radiol, 2014, 83(10), 1771-177743. 王水,张保康,赵同生, Hassab 手术后肝硬变大鼠动脉血酮体比改变的意义, 南京医学院学报, 1991, (04), 302-30544. 王水,刘训良,陆辉,孔连宝, B 型 Niemann-Pick 病脾脏切除一例, 南京医学院学报, 1993, (02), 18645. 王水,沈亚勋, 老年原发性胆囊癌误诊 27 例分析, 南京医科大学学报, 1997, (02), 7746. 王水,查小明,武正炎, 原发性甲状旁腺机能亢进症术后低钙血症及其处理, 江苏医药, 2001, (03), 18047. 王水,查小明, 三苯氧胺对绝经后乳腺癌患者子宫和卵巢的影响, 南京医科大学学报, 2001, (04), 320-32148. 王水,林红军,查小明, 三苯氧胺对乳腺癌患者子宫卵巢的影响, 中国肿瘤临床, 2001, (05), 389-39049. 王水,武正炎,范萍,查小明, 血管内皮细胞生长因子在乳腺癌组织中的表达及其生物学意义, 南京医科大学学报, 2001, (06), 503-50550. 王水,查小明,范萍,武正炎, 乳腺癌染料法前哨淋巴结活检的临床意义, 中华肿瘤杂志, 2002, (02), 9051. 王水,刘晓,刘晓安,查小明,符德元,甄林林,范萍,武正炎,乳腺癌前哨淋巴结活检染料和手术时机选择,肿瘤学杂志, 2002, (06), 332-33552. 王水,刘晓安,武正炎,影响染料法乳腺癌前哨淋巴结活检成功率的一些因素外科理论与实践, 2004, (02), 107-10953. 王水,范萍,武正炎,乳腺癌前哨淋巴结淋巴细胞亚群的初步研究,中华肿瘤杂志, 2004, (04), 31-3354. 王水,刘晓安,赵燕超,武正炎,采用异硫蓝和美蓝行乳腺癌前哨淋巴结活检的临床价值,江苏医药, 2004, (07), 506-50755. 王水,范萍,刘晓安,武正炎,乳腺癌骨髓和前哨淋巴结微小转移检测的临床研究,中华普通外科杂志, 2004, (09), 54-5556. 耿强,王水,范萍,刘晓安,郑伟,乳腺癌前哨淋巴结和骨髓和外周血微小转移的联合检测,肿瘤防治杂志, 2005, (02), 85-8857. 徐鲲,刘晓安,王水,张小林,陆辉,乳腺癌前哨淋巴结活检染料选择的实验研究,南京医科大学学报(自然科学版), 2005, (03), 201-20358. 徐鲲,刘晓安,王水,王振中,乳腺癌前哨淋巴结活检常用染料效果实验研究,现代肿瘤医学, 2005, (04), 433-43559. 徐鲲,刘晓安,王水,周青,耿强,中华墨汁作为乳腺癌前哨淋巴结活检示踪剂的实验研究,肿瘤防治杂志, 2005, (05), 321-32460.王水,刘晓安,早期乳腺癌治疗中前哨淋巴结活检应用价值探讨,中国实用外科杂志, 2006, (04), 312-31461. 王水,赵佳,刘晓,甲状腺炎合并甲状腺功能亢进的手术治疗,中国实用外科杂志, 2006, (07), 503-50562. 王水,刘晓安,赵佳,耿强,刘力嘉,染料法乳腺癌前哨淋巴通道的研究,中华外科杂志, 2006, (11), 748-75063. 曹勤洪,王水,徐鲲 ,刘晓安,刘力嘉,中华墨汁作为前哨淋巴结活检新型示踪剂的实验研究,The Chinese-German Journal of Clinical Oncology, 2006, (01), 36-3964. 郝汉霞,王水,刘力嘉,范萍,杜青,张伟民,王聪, 乳腺癌肿瘤相关巨噬细胞中 HIF-2α/EPAS1 表达与血管生成的关系,中华肿瘤防治杂志, 2006, (03), 169-17265. 王锋,王水,陆辉,郑伟,孙琳, 乳腺癌组织中 p21~(WAF1)、CDK4 的表达及临床意义,江苏医药, 2006, (06), 520-52266. 赵佳,刘晓安,王水,刘力嘉 免疫组织化学和RT-PCR法检测乳腺癌骨髓和前哨淋巴结微小转移的临床研究现代肿瘤医学, 2006, (07), 813-81667. 邹汉青,王水,林红军,张智弘,刘晓安,彭晓静, 活体兔肌肉组织微波固化后病理学变化的实验研究 中华肿瘤防治杂志, 2006, (07), 489-49168. 凌立君,魏惠华,曾莉,刘晓安,王凤良,赵佳,贾晓斌,王水,盐酸川芎嗪微囊的制备及质量控制,中成药, 2006, (11), 1692-169369. 赵佳,王水,刘晓安,刘力嘉,IHC 和 PCR 法联合检测乳腺癌前哨淋巴结的微转移,中华肿瘤防治杂志, 2006, (16), 1229-123170. 王水,刘晓安,赵佳,耿强,刘力嘉,联合法观察前哨淋巴通道的临床研究,中华乳腺病杂志(电子版), 2007, (01), 35-3871. 王锋,王水,宣卓琦,孙琳,陆辉,乳腺癌细胞中 p53、C-erbB-2、p21~(WAF1)和 CDK4 的表达及临床意义, 南京医科大学学报(自然科学版), 2007, (01), 70-7372. 凌立君,赵佳,王水,刘晓安,核素法前哨淋巴通道的研究,中华乳腺病杂志(电子版), 2007, (02), 16-1873. 王锋,夏添松,夏建国,陈国玉,王水胃肠道间质肿瘤伴肝转移 18 例临床分析,临床肿瘤学杂志, 2007, (03), 191-19374. 许健,王水,许立生,杜青,刘晓安,乳腺癌细胞系中肿瘤干细胞相关亚群初步研究,南京医科大学学报(自然科学版), 2007, (04), 350-35575. 谢锡驹,王水,郑伟,查小明,乳腺癌患者血清 IGF-Ⅱ水平及其在肿瘤组织中的表达,江苏医药, 2007, (07), 672-67476. 王凤良,凌立君,沈恩超,刘晓安,王水,血清 Tracp5b 诊断乳腺癌骨转移的临床价值,南京医科大学学报(自然科学版), 2007, (07), 702-70577. 许健; 王水;许立生,杜青,刘晓安,赵佳,雌激素对 MCF-7 中肿瘤干细胞相关亚群影响的实验研究,中华肿瘤防治杂志, 2007, (09), 650-65278. 陈龙舟,赵佳,王凤良,凌立君,刘晓安,王水 淋巴结阴性乳腺癌骨髓微小转移联合检测的临床研究,南京医科大学学报(自然科学版), 2007, (09), 939-94279. 许立生,王水,许健,杜青,无血清悬浮法培养MCF-7细胞系并筛选该细胞系中肿瘤干细胞相关亚群的初步研究,中华肿瘤防治杂志, 2007, (10), 736-74080. 谢晖,曹勤洪,夏锴,刘晓安,王水乳腺癌组织 APRG1 基因表达的初步研究,江苏医药, 2007, (10), 1006-100881. 刘月仙,夏添松,李俊芳,王水,共刺激分子 B7-2 基因转染抑制乳腺癌生长和转移的实验研究,南京医科大学学报(自然科学版), 2007, (11), 1228-123282. 孙蓉,凌立君,刘晓安,赵佳,耿强,王水,联合法前哨淋巴通道的临床研究,中华肿瘤防治杂志, 2007, (17), 1328-133083. 王凤良,王水,凌立君,沈恩超,刘晓安,血清 Tracp5b 诊断乳腺癌骨转移及评价双膦酸盐治疗疗效的临床意义,中华肿瘤防治杂志, 2007, (21), 1628-163184. 王水,赵佳,乳腺癌前哨淋巴结活检的研究进展,中华普外科手术学杂志(电子版), 2008, (02), 147-15285. 王水,赵佳,早期乳腺癌前哨淋巴结活检术的进展,中华乳腺病杂志(电子版), 2008, (03), 255-26186. 王水,重视老年甲状腺疾病诊断与治疗,实用老年医学, 2008, (04), 243-24487. 李昌文,刘晓安,林红军,章宜芬,王水冷循环微波固化家猪乳腺实验研,南京医科大学学报(自然科学版), 2008, (01), 16-1988. 沈恩超,凌立君,王水,刘晓安,朱海清,李凤山,SDF-1、CXCR4 在乳腺癌组织及转移淋巴结中的表达及其意义,江苏医药, 2008, (04), 367-36989. 钱超,夏锴,张静静,杜青,刘晓安,王水缺氧条件下雷帕霉素抗人乳腺癌 MCF-7 细胞增殖作用的研究,南京医科大学学报(自然科学版), 2008, (06), 742-74690. 高泽俊,袁小青,沈捷,马向华,王水,MAPKs通路在Leptin促人乳腺癌细胞系增殖中的机制研究,南京医科大学学报(自然科学版), 2008, (11), 1410-141491.刘学敏,王水,夏添松,王珏,刘晓安 慢病毒转染绿色荧光蛋白对乳腺癌细胞系肿瘤干细胞相关亚群的影响, 中国组织工程研究与临床康复, 2008, (38), 7507-751092.王水,乳管镜技术临床应用及其诊疗价值,中国实用外科杂志, 2009, (03), 261-26393. 顾则娟,王水,张镇静,王荣,谢晓峰,朱琳,戴晓冬,叶桂华,林征,方小平, 殷蓉,舒心病房护理运行模式的建立与应用,中华护理杂志, 2009, (10), 929-93194. 宋兵,沈历宗,徐皓,黄华兴,肇毅,史京萍,王水外科实习生出科量化考核结果的研究分析与思考,临床和实验医学杂志, 2010, (05), 353-35595. 傅卫,王水,杜青,肇毅,查小明,刘晓安,巢式 RT-PCR 检测乳腺癌外周血循环肿瘤细胞的临床价值,南京医科大学学报(自然科学版), 2010, (06), 818-82196. 蒋超,丁强,王水,杨海伟,张炜明,FISH 检测乳腺癌石蜡组织切片的酶消化时间的探究,现代生物医学进展, 2010, (15), 2907-290997. 陈翔,王水,乳腺癌前哨淋巴结活检研究现状及进展,中国普外基础与临床杂志, 2011, (01), 98-10298. 黄华兴,沈历宗,肇毅,凌立君,史京萍,徐皓,吴蔚,王水,PBL与LBL相结合在基础外科学实践教学中的应用,南京医科大学学报(社会科学版), 2011, (03), 227-22999. 崔一尧,王水,临床推广乳腺癌前哨淋巴结活检技术的影响因素分析,中国医学伦理学, 2011, (05), 611-613100. 黄华兴,沈历宗,肇毅,凌立君,史京萍,徐皓,吴蔚,王水,外科学基础考试模式的改革与探讨,南京医科大学学报(社会科学版), 2011, (06), 488-490101. 崔一尧,王水,前哨淋巴结活检在乳腺癌治疗中的意义,医学与哲学(临床决策论坛版), 2011, (08), 41-42102. 史京萍,夏添松,丁强,刘晓安,査小明,王水,CpG 寡核苷酸促进 Toll 样受体 9 的表达增强乳腺癌细胞侵袭南京医科大学学报(自然科学版), 2011, (08), 1161-1163103. 崇梅红,王水,乳腺癌血行微小转移研究进展,中华临床医师杂志(电子版), 2011, (10), 2970-2974104. 崇梅红,王水,乳腺癌淋巴结微小转移研究进展,中华临床医师杂志(电子版), 2011, (11), 3254-3257105. 许立生,王水,黄中晶,沈恩超,钱超,人生长激素对乳腺癌细胞增殖和凋亡的影响,天津医药, 2011, (12), 1141-1143106. 马晶晶,王水,郑明洁,缪苏宇,徐露,王珏,乳腺癌细胞株两种原代培养方法的比较,江苏医药, 2011, (16), 1865-1867107. 刘钊,王水,孕激素及雌激素对乳腺癌细胞 RANKL 及其受体的调节作用,江苏医药, 2011, (16), 1874-1876108. 许立生,王水,黄中晶,沈恩超,钱超,CD44~+/CD24~(-/low)/ABCG2~-乳腺癌干细胞与临床治疗及预后的关系,实用医学杂志, 2011, (21), 3877-3879109. 崇梅红,王水,查小明,刘晓安,凌立君,肇毅,实时荧光定量PCR法检测乳腺癌外周血循环肿瘤细胞的临床价值,南京医科大学学报(自然科学版), 2012, (02), 220-225110. 赵奕华,李鑫,王水,王虹,公立医院改革背景下医用耗材管理的困难与对策,中国医疗设备, 2012, (11), 100-102111.周茜,王水,钱宁,刘小丰,陆澄,骨桥蛋白在乳腺癌组织中的表达及其与临床病理和 p53 的关系,江苏医药, 2012, (20), 2387-2389112. 许立生,王水,黄中晶,赵志泓,沈恩超,钱超,ER、PR、VEGF、CA15-3、CA125 和 CEA 水平在乳腺癌预后判断中的临床意义,实用医学杂志, 2012, (21), 3565-3567113. 张桂普,王水,郑明洁,王珏,“人源性”乳腺微环境促进人源性乳腺癌细胞发生上皮细胞间充质转化,江苏医药, 2012, (22), 2652-2654114. 王水,刘钊,乳腺神经内分泌癌,中国实用外科杂志, 2013, (03), 238-240115. 姚秀蕾,肇毅,姚志勇,王水,超声引导粗针活检在触诊阴性乳腺肿瘤诊断中的临床意义,中国医药导报, 2013, (02), 41-43116. 黄华兴,张永杰,沈历宗,肇毅,王水,整合外科与解剖教学,架构临床与基础桥梁,南京医科大学学报(社会科学版), 2013, (03), 273-275117.周悦,黄华兴,骆金华,陈亮,王水,胸腔镜技术应用以提高胸心外科教学效果的研究与实践,南京医科大学学报(社会科学版), 2013, (05), 472-473118. 杨小冬,黄华兴,黄平,沈历宗,王水,浅谈外科学基础实践教学师资的选拔与培养,南京医科大学学报(社会科学版), 2013, (06), 564-566119. 朱倩男,夏添松,凌立君,王水,乳腺癌术后上肢淋巴水肿发生机制及预防进展,中华乳腺病杂志(电子版), 2013, (06), 442-445120. 王若曦,王水,乳腺癌 ER、PR 及 HER-2 在原发灶和转移灶之间表达关系的研究进展,江苏医药, 2013, (08), 958-961121.吴丹,刘晓安,查小明,王水,乳腺淋巴瘤 12 例临床分析,江苏医药, 2013, (14), 1660-1662122.吴蔚,沈历宗,肇毅,凌立君,史京萍,黄华兴,王水,积极心理学在外科学总论实验教学中的应用,中国医学创新, 2013, (34), 141-142123.徐海萍,王娟,孙杰,王珏,王水,查小明,鲁婷婷,孙茹萍,杨晓文,优化乳腺癌健康宣教的探讨,江苏医药, 2014, (05), 604-606
专业领域:乳腺癌杂志国家:美国投稿系统:是否收版面费:是分享的经验或教训:1、投稿20天左右可以发信问一问,我投的时候用了差不多一个晚上,网络太拥挤太慢。所以误以为结束了,但是实际上没有递交成功。白白的耽误了两个月。我的催问信 ,供参考:Dear Dr. Lewis A Chodosh ,(该杂志主编)We submitted a paper entitled " *** " (Manuscript ID: 5340265452290289) on October 15, 2008. About 50 days has past, but the manuscript is still under review. I am quite anxious for the current status regarding our paper. And how long dose the review progress last, usually?When can we receive your decision letter?Looking forward to yours reply!Kind sincerely,***2、投这个杂志主要是身边有师兄投了,而且中了。该杂志偏重于临床,乳腺癌患者或荷瘤动物研究,可能比细胞系研究有优势。当然文章要有新意,那就不必说了。3、杂志的速度不算太快。你若急着赶时间,建议稿投其他速度快的杂志。
分类: 教育/科学 >> 外语学习 问题描述: Do you think our life is full of causes of cancer?why? 200-300词左右,请多举例,谢谢! 解析: I think there are many of ways that can cause cancer in our daily life. Cancer has many causesThere are about 200 different types of cancer affecting all the different body tissues. What affects one body tissue may not affect another. For example, tobacco *** oke that you breathe in may help to cause lung cancer. Over exposing your skin to the sun could give you a melanoma on your leg. But the sun won't give you lung cancer and *** oking won't give you melanoma. Apart from infectious diseases, most illnesses are 'multifactorial'. Cancer is no exception. Multifactorial means that there are many factors involved. In other words, there is no single cause for any one type of cancer. Carcinogens A 'carcinogen' is something that can help to cause cancer. Tobacco *** oke is a powerful carcinogen. But not everyone who *** okes gets lung cancer. So there must be other factors at work. Age Most types of cancer bee more mon as we get older. This is because the changes that cause a cell to bee cancerous in the first place take a long time to develop. There have to be a number of changes to the genes within a cell before it turns into a cancer cell. The changes can happen by accident when the cell is dividing. Or they can happen because the cell has been damaged by carcinogens and the damage is then passed on to future 'daughter' cells when that cell divides. The longer we live, the more time there is for us to accumulate these geic mistakes in our cells. Geic make up There have to be a number of geic mutations within a cell before it bees cancerous. Sometimes we are born with one of these mutations already. This does not mean we will get cancer. But with one mutation from the outset, it makes it more likely statistically that we will. Doctors call this 'geic predisposition'. The BRCA1 and BRCA2 breast cancer genes are examples of geic predisposition. Women who carry one of these faulty genes have a higher chance of developing breast cancer than women who do not. The BRCA genes are good examples for another reason. Most women with breast cancer do not have a mutated BRCA1 or BRCA 2 gene. Less than 5% of all breast cancer is due to these genes. So although women with one of these genes are individually more likely to get breast cancer, most breast cancer is not caused by a high risk inherited gene fault. This is true of other mon cancers where some people have a geic predisposition, for example colon (large bowel) cancer. Researchers are looking at the genes of people with cancer in a study called SEARCH. They also hope to find out more about how other factors might interact with genes to increase the risk of cancer. Information about this study is on our clinical trials database. Either follow the link or click on the blue button to the left of any CancerHelp UK screen. Then type SEARCH into the Free Text Search box. Please note that you cannot volunteer for this study. The immune system People who have problems with their immune systems are more likely to get some forms of cancer. This group includes people who Have had an transplants and take drugs to suppress their immune systems to stop an rejection Have AIDS Are born with rare medical syndromes which affect their immunity The kinds of extra cancers that affect these groups of people fall into o, overlapping groups Cancers that are caused by viruses, such as cervical cancer or some lymphomas Lymphomas Chronic infections or transplanted ans can continually stimulate cells to divide. This continual cell division means that immune cells are more likely to acquire mutations and develop into lymphomas. Diet Cancer experts estimate that changes to our diet could prevent about one in three cancer deaths in the UK. In the western world, many of us eat too many animal fats and not enough fresh fruit and vegetables. This type of diet is known to increase your risk of cancer. But how exactly we should alter our diets is not clear. There is more about this in the page on diet causing cancer. Sometimes foods or food additives are blamed for directly causing cancer and described as 'carcinogenic'. This is often a distortion of the truth. Sometimes a food is found to contain a substance that can cause cancer but in such *** all amounts that we could never eat enough of it to do any harm. And some additives may actually protect us. There is more about food additives in the page on diet causing cancer. Day to day environment By this we mean what is around you each day that may help to cause cancer. This could include Tobacco *** oke The sun Natural and man made radiation Work place hazards A *** estos Some of these are avoidable and some aren't. Most are only contributing factors to causing cancers - part of the jigsaw puzzle that scientists are still trying to put together. There is more about this in the page on causes of cancer in the environment . Viruses Viruses can help to cause some cancers. But this does not mean that these cancers can be caught like an infection. What happens is that the virus can cause geic changes in cells that make them more likely to bee cancerous. These cancers and viruses are linked Cervical cancer and the genital wart virus, HPV Primary liver cancer and the Hepatitis B virus T cell leukaemia in *** s and the Human T cell leukaemia virus There will be people with primary liver cancer and with T cell leukaemia who haven't had the related virus. But infection may increase their risk of getting that particular cancer. With cervical cancer, scientists now believe that everyone with an invasive cervical cancer will have had an HPV infection beforehand. Many people can be infected with a cancer-causing virus, and never get cancer. The virus only causes cancer in certain situations. Many women get a high risk HPV infection, but never develop cervical cancer. Another example is Epstein-Barr virus (EBV). These are some facts about this mon virus Most people are infected with EBV People who catch it late get glandular fever but this does not cause cancer In sub-Saharan Africa, EBV infection and repeated attacks of malaria together cause a cancer called Burkitt's lymphoma that affects children In China, EBV infection (together with other unknown factors) causes naso-pharyngeal cancer In AIDs patients and transplant patients EBV can cause lymphoma In the UK, about 4 out of 10 cases of Hodgkin's disease seem to be related to EBV infection 可以适当删减
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