发布时间：
这种情况我遇到过，那位编辑是个大好人。当时也显示review了，不过应该是内审，一周后拒稿的。拒稿信里说明了他们期刊不收录我这个方向的文章，但是他觉得sci论文本身挺好的就是存在一些语法上的瑕疵，于是帮我从编辑的角度手工润色了一遍附在拒稿信里，建议我去投另外一个期刊
可以啊
审稿时间与被拒无关。sci论文投稿录用率主要和论文内容质量，投稿期刊，以及投稿操作流程，经验的积累是有关的，与审稿时间长短无关。
一般SCI期刊的重复率要求是，整体20%以下，单篇3%以下。所以百分之十八是可以的。重复率等级低重复率(小于10%)这类论文中所检出的相似率一般都是 一些来源相对分散的匹配文本或常见短语 。单个匹配来源同时很低，仅仅占1%-3%的相似率。如果你的文章重复率在10%，审稿人绝对 不会因为重复率问题 予以拒稿。个人认为作为理工科论文，特别是应用类型的话，这个还是比较难以做到的。中等重复率(10%-50%之间)这个范围区间是 大部分研究者的论文所在的区间，属于适中的百分比 ，此时就需要具体情况具体分析了。这类论文可能是由于多处1%-3%的来源组成，或者单个来源比较高。作者需要对文章进行简单的检查判断：如果有单处来源的重复率高于5%，那么就请注意了，需要对这些部分进行改写并且深度润色。其实不仅仅是单篇高于10% ，如果重复的地方多了，积少成多也会有20%左右，特别是科技类论文。例如：我的一篇论文单篇重率不大于2%，但是各个小的句子累积起来有76处之多 很大程度因为我做的研究和别人的论述相同，特别是深度学习中的术语和公式！
修回后还是有可能被拒的。
�皇嵌晕颐钦庑┬〉ノ坏娜司涂嗔耍�蛭�悴蝗鲜端�牵�膊豢赡苁撬�堑难��Ｄ阒挥小⒅荒芡ü�岣咦约何恼轮柿坎鸥�谢�帷；褂芯褪锹���省⑿��,但目前因为杂志生存的原因，较之以前速度可能也都快起来了，也甚至还有些只要给钱就收的杂志，类似于西太平洋大学似的，这是卖文凭，人家就是卖文章的，当然我想大部分杂志还是能够“客观、公正”进行稿源选择吧，但愿吧。 那SCI杂志又是如何审稿呢，相反应该更“客观”，至少不会歧视你是来自无名的单位，就受邀于几个杂志作为其审稿人来说，绝大部分都采用同行审稿（peer-review），也即邀请你审稿的文章多半是与被邀请人所作的研究相似，这就不存在一定是所谓的“牛人”来审了。与你的职称完全没有关系，不是说教授就有资格，而初级就没有资格被SCI杂志邀请审稿杂志社一般都会通过你发表文章找到你的email（我想应该是这样的，所以通讯更牛叉啊，否在人家不会邀请到你的） 当然你也可以拒绝，因为毕竟不是所有的文章我们都有能力去审的，至少我还没那水平，也曾拒绝过一篇关于基因分析的（虽然了解一些，但很难把握）如你接受后，则会自动连接到审稿人页面（如下），除了author centre外，同时也有reviewer centre的页而一般如果没有被邀请审稿，通常进入作者系统后，只会有anthor centre接下来就是审稿了，这篇文章是国内某F教授为通讯的一篇关于血管紧张素在皮肤中作用的综述，毕竟人家在国内还是相当的啊，也是顺便学习了一下，可一下载稿件一看，怎么看怎么不像综述，充其量是个mini review，四个段落，1198个字，26篇参考文献。既然杂志社邀请我审稿，也综述的我所做过的研究，却没引用我的文章，有点“失落”，更重要的是更多文献他们也没有引用，而是引用他们自己的“中文”文章,但还是认真的“分析”了此文。给出审稿意见如下：The review by *** et addresses the interesting and novel topic of the renin-angiotensin-system (RAS), which was originally described as a cardiovascular endocrine system, in skin physiology and Only in recent years, the cutaneous RAS has become an area of scientific interest, and the number of related publications is increasing from year to Therefore, it is indeed time for an article which reviews the existing literature up to However, this review has a number of 中国人语言每次都被审稿人发飙，我的也是，至少部分文章投出去审稿人也是要让我改进，唔，每次让我找个母语的人帮修改，我滴个汗，俺们中国人哪有以说英语母语的，索性每次都“忽悠”过去了，当然也许只是些小杂志的原因吧，语言真的需要提高、再提高。同样该综述，我也提出了我的部分语言意见 - Language editing is - The number of publications dealing with the RAS in skin is still not very Therefore, there is no need to focus this review article on the AT2-receptor (for which data are even more limited) Instead, this review should consider all published data about the cutaneous RAS - Many publications are For example: �6�1Min et , Endocrinology 2004 �6�1Nakai et , J Dermatol Sci 2008 �6�1Rompe et , Hypertension 2010 �6�1Steckelings et , (Exp Dermatol 2004) �6�1Yevdokimova et , J Dermatol Sci 2007 �6�1Morihara et , J Am Acad Dermatol 2006 �6�1Yahata et , J Biol Chem 2006 �6�1Takeda et , Am J Pathol 2004 并逐部分给予我的意见：Abstract:- In the case of AT1R-blockade, AT2R unmasking may indeed be important, but blockade of the AT1R thus interrupting AT1R-mediated actions of Ang II, is at least as The respective passage in the abstract is Introduction: - 3, line 13: “disorders of RAS”: A “disorder” of the RAS has so far only been described for scleroderma (not saying whether the deregulated RAS is a primary cause or only a secondary phenomenon) It is indeed likely that the RAS is deregulated in the other mentioned dermatoses as well, but this is pure speculation and should be discussed as - 3, line 19: “existence of RAS in skin”: References 2 and 3 demonstrate only the existence of receptors, but not of the whole RAS in Adequate references would be: Steckelings et , Exp Dermatol or Philips MI et , In: Saavedra J M, Timmermans P M W M, Angiotensin New York: Plenum Press, 1994: 377– - 3, line 20: “It has been documented…”: It is correct that AT2R upregulation has been demonstrated in skin, and it is also correct that Ang II has been shown to accelerate cutaneous wound However, it has never been shown that acceleration of wound healing by Ang II is mediated by the AT2R In fact, this is rather unlikely, since the AT2R acts anti- - Chapter II 1: Physiological receptor expression should be addressed prior to receptor expression in - p4, line 5 from bottom: “Ang II either…” Please add - chapter II 2: The high expression of Ang II receptors during foetal life indeed suggests a role in However, Ang II receptor knockout mice show no severe developmental deficits, in particular not in Furthermore, there are almost no data about what specifically the role of the AT2R in development may This should be - chapter II 3: This chapter is much too For example, the description of deregulated receptor expression in some dermatoses by Takeda and Kondo (Am J Pathol 2001, Br J Dermatol 2001 and 2002) has not been This chapter may further be the place for some speculations (based on data from non-cutaneous tissues) in which dermatoses the RAS may be - page 5, line 5: “Kawaguchi et al …in SSc fibroblasts, suggesting that… “: This is not a logical What is the causal link between AT2R in SSc fibroblasts and excessive ECM production? Furthermore, expression of AT2R has been shown by several authors for normal - page 5, line 12: Steckelings is a woman (“her” colleagues) - page 5, last section: The impact of AT2R expression on immune cells and of AT2R effects on vascularisation and neuroregeneration with regard to wound healing is not sufficiently - 6, 2 lines from bottom: “…restoring normality not only in the CV system but also in many tissues, such as ” Please provide a reference for the statement that the AT2R has been shown to restore normality in 最后提交时杂志社会有一个勾选表，该文被我拒了该文编辑在结合另一个审稿人意见的情况下还是reject此文了，从投稿到最后给出decision约6个星期，应该说是正常速度了。有意思的是中途，编辑发信催审稿了，估计是作者急着想知道结果，可以理解，想想之前的我们也何尝不是啊，每天都不停得刷屏，也写过催稿信，还以为没有用，甚至有时候也不“敢”写，因为害怕是否会有“反”作用，看来某些时候写信催催也还是可以的。总之，审稿也未必是件好差事，不过倒是可以知己知彼，可以站在审稿人的角度去思考我们自己在写文章的时候应该注意什么，别人文章的有哪些优点、缺点，我们都可以好好去总结，同时我们也获得与最新研究领域的接触也为以后研究，能够写作提供更多的思路。
区别主要是内容和主题上的区别。如下：如果是SCi拒稿的话通常情况下是说明你的稿子不够新颖不符合他们的主题或者是他们最近想采用的主题和你的所写的内容不太符合，但是这不证明你的稿子写的不好，只是不符合sci的主题而已。如果是拒稿重投，那就说明你的内容可能还没有达到他们的要求，就例如像是觉得你的数据不真实或者是些许欠缺，再或者说是有的部分言论前后没有衔接好，这就说明可能是你文章的内容的问题，需要下次去进行修改。
建议你的手稿适合发表在《科学进步》杂志上，这是一份多学科期刊，旨在为所有科学探索领域的研究人员提供一个平台。你的论文已经在’彻底审查第一次投稿期刊因此不需要进一步审查。”
�皇嵌晕颐钦庑┬〉ノ坏娜司涂嗔耍�蛭�悴蝗鲜端�牵�膊豢赡苁撬�堑难��Ｄ阒挥小⒅荒芡ü�岣咦约何恼轮柿坎鸥�谢�帷；褂芯褪锹���省⑿��,但目前因为杂志生存的原因，较之以前速度可能也都快起来了，也甚至还有些只要给钱就收的杂志，类似于西太平洋大学似的，这是卖文凭，人家就是卖文章的，当然我想大部分杂志还是能够“客观、公正”进行稿源选择吧，但愿吧。 那SCI杂志又是如何审稿呢，相反应该更“客观”，至少不会歧视你是来自无名的单位，就受邀于几个杂志作为其审稿人来说，绝大部分都采用同行审稿（peer-review），也即邀请你审稿的文章多半是与被邀请人所作的研究相似，这就不存在一定是所谓的“牛人”来审了。与你的职称完全没有关系，不是说教授就有资格，而初级就没有资格被SCI杂志邀请审稿杂志社一般都会通过你发表文章找到你的email（我想应该是这样的，所以通讯更牛叉啊，否在人家不会邀请到你的） 当然你也可以拒绝，因为毕竟不是所有的文章我们都有能力去审的，至少我还没那水平，也曾拒绝过一篇关于基因分析的（虽然了解一些，但很难把握）如你接受后，则会自动连接到审稿人页面（如下），除了author centre外，同时也有reviewer centre的页而一般如果没有被邀请审稿，通常进入作者系统后，只会有anthor centre接下来就是审稿了，这篇文章是国内某F教授为通讯的一篇关于血管紧张素在皮肤中作用的综述，毕竟人家在国内还是相当的啊，也是顺便学习了一下，可一下载稿件一看，怎么看怎么不像综述，充其量是个mini review，四个段落，1198个字，26篇参考文献。既然杂志社邀请我审稿，也综述的我所做过的研究，却没引用我的文章，有点“失落”，更重要的是更多文献他们也没有引用，而是引用他们自己的“中文”文章,但还是认真的“分析”了此文。给出审稿意见如下：The review by *** et addresses the interesting and novel topic of the renin-angiotensin-system (RAS), which was originally described as a cardiovascular endocrine system, in skin physiology and Only in recent years, the cutaneous RAS has become an area of scientific interest, and the number of related publications is increasing from year to Therefore, it is indeed time for an article which reviews the existing literature up to However, this review has a number of 中国人语言每次都被审稿人发飙，我的也是，至少部分文章投出去审稿人也是要让我改进，唔，每次让我找个母语的人帮修改，我滴个汗，俺们中国人哪有以说英语母语的，索性每次都“忽悠”过去了，当然也许只是些小杂志的原因吧，语言真的需要提高、再提高。同样该综述，我也提出了我的部分语言意见 - Language editing is - The number of publications dealing with the RAS in skin is still not very Therefore, there is no need to focus this review article on the AT2-receptor (for which data are even more limited) Instead, this review should consider all published data about the cutaneous RAS - Many publications are For example: �6�1Min et , Endocrinology 2004 �6�1Nakai et , J Dermatol Sci 2008 �6�1Rompe et , Hypertension 2010 �6�1Steckelings et , (Exp Dermatol 2004) �6�1Yevdokimova et , J Dermatol Sci 2007 �6�1Morihara et , J Am Acad Dermatol 2006 �6�1Yahata et , J Biol Chem 2006 �6�1Takeda et , Am J Pathol 2004 并逐部分给予我的意见：Abstract:- In the case of AT1R-blockade, AT2R unmasking may indeed be important, but blockade of the AT1R thus interrupting AT1R-mediated actions of Ang II, is at least as The respective passage in the abstract is Introduction: - 3, line 13: “disorders of RAS”: A “disorder” of the RAS has so far only been described for scleroderma (not saying whether the deregulated RAS is a primary cause or only a secondary phenomenon) It is indeed likely that the RAS is deregulated in the other mentioned dermatoses as well, but this is pure speculation and should be discussed as - 3, line 19: “existence of RAS in skin”: References 2 and 3 demonstrate only the existence of receptors, but not of the whole RAS in Adequate references would be: Steckelings et , Exp Dermatol or Philips MI et , In: Saavedra J M, Timmermans P M W M, Angiotensin New York: Plenum Press, 1994: 377– - 3, line 20: “It has been documented…”: It is correct that AT2R upregulation has been demonstrated in skin, and it is also correct that Ang II has been shown to accelerate cutaneous wound However, it has never been shown that acceleration of wound healing by Ang II is mediated by the AT2R In fact, this is rather unlikely, since the AT2R acts anti- - Chapter II 1: Physiological receptor expression should be addressed prior to receptor expression in - p4, line 5 from bottom: “Ang II either…” Please add - chapter II 2: The high expression of Ang II receptors during foetal life indeed suggests a role in However, Ang II receptor knockout mice show no severe developmental deficits, in particular not in Furthermore, there are almost no data about what specifically the role of the AT2R in development may This should be - chapter II 3: This chapter is much too For example, the description of deregulated receptor expression in some dermatoses by Takeda and Kondo (Am J Pathol 2001, Br J Dermatol 2001 and 2002) has not been This chapter may further be the place for some speculations (based on data from non-cutaneous tissues) in which dermatoses the RAS may be - page 5, line 5: “Kawaguchi et al …in SSc fibroblasts, suggesting that… “: This is not a logical What is the causal link between AT2R in SSc fibroblasts and excessive ECM production? Furthermore, expression of AT2R has been shown by several authors for normal - page 5, line 12: Steckelings is a woman (“her” colleagues) - page 5, last section: The impact of AT2R expression on immune cells and of AT2R effects on vascularisation and neuroregeneration with regard to wound healing is not sufficiently - 6, 2 lines from bottom: “…restoring normality not only in the CV system but also in many tissues, such as ” Please provide a reference for the statement that the AT2R has been shown to restore normality in 最后提交时杂志社会有一个勾选表，该文被我拒了该文编辑在结合另一个审稿人意见的情况下还是reject此文了，从投稿到最后给出decision约6个星期，应该说是正常速度了。有意思的是中途，编辑发信催审稿了，估计是作者急着想知道结果，可以理解，想想之前的我们也何尝不是啊，每天都不停得刷屏，也写过催稿信，还以为没有用，甚至有时候也不“敢”写，因为害怕是否会有“反”作用，看来某些时候写信催催也还是可以的。总之，审稿也未必是件好差事，不过倒是可以知己知彼，可以站在审稿人的角度去思考我们自己在写文章的时候应该注意什么，别人文章的有哪些优点、缺点，我们都可以好好去总结，同时我们也获得与最新研究领域的接触也为以后研究，能够写作提供更多的思路。